Activation of ATM depends on chromatin interactions occurring before induction of DNA damage

Keyphrases

• DNA Damage 100%
• Chromatin Interactions 100%
• Ataxia Telangiectasia mutated 100%
• Double-strand Breaks 50%
• HMGN1 50%
• Chromatin 30%
• Ionizing Radiation 20%
• Steady State 10%
• Binding Protein 10%
• Radiation Therapy 10%
• Radiation Effects 10%
• Structural Change 10%
• Protein Kinase 10%
• Histone Modification 10%
• Ablation 10%
• Cellular Response 10%
• Genome Stability 10%
• Chromosome Rearrangement 10%
• H3K14 10%
• Nucleosome Binding 10%
• Histone H3 10%
• Acetylation 10%
• Signaling Network 10%
• Chromosomal DNA 10%
• Histone Deacetylase Inhibitor (HDACi) 10%
• Intranuclear 10%
• Autophosphorylation 10%
• Chromatin Architecture 10%

Biochemistry, Genetics and Molecular Biology

• Ataxia Telangiectasia Mutated 100%
• DNA Damage 100%
• Mediator 10%
• Histone Deacetylase 10%
• Steady State 10%
• Binding Protein 10%
• Genome Instability 10%
• Protein Kinases 10%
• Autophosphorylation 10%
• Histone Modification 10%
• Acetylation 10%
• Nucleosome 10%
• Histone H3 10%