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Disrupting the MAD2L2-Rev1 Complex Enhances Cell Death upon DNA Damage
Nomi Pernicone
, Maria Elias
, Itay Onn
,
Dror Tobi
,
Tamar Listovsky
Department of Computer Science
Department of Molecular Biology
Research output
:
Contribution to journal
›
Article
›
peer-review
10
Scopus citations
Overview
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Dive into the research topics of 'Disrupting the MAD2L2-Rev1 Complex Enhances Cell Death upon DNA Damage'. Together they form a unique fingerprint.
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Keyphrases
Cell Death
100%
DNA Damage
100%
MAD2L2
100%
Translesion Synthesis
100%
Chemotherapy
33%
Cisplatin
33%
Small Molecules
16%
Tumor Formation
16%
Promising Strategies
16%
Translesion DNA Polymerase
16%
Regulatory Complexity
16%
REV7
16%
Acquired Drug Resistance
16%
Secondary Malignancy
16%
Underlining
16%
Cisplatin Resistance
16%
Lung Cancer Cell Lines
16%
Polymerase
16%
DNA Polymerase
16%
Chemotherapy Resistance
16%
Chemotherapy Treatment
16%
Drug Resistance
16%
Single-strand Gap
16%
Secondary Tumor
16%
Synthesis Pathway
16%
DNA Damage Tolerance
16%
Gap Filling
16%
Chemotherapy Agents
16%
Treatment Lines
16%
Intrinsic Drug Resistance
16%
DNA-damaging
16%
Central Protein
16%
Medicine and Dentistry
Cell Death
100%
DNA Damage
100%
Chemotherapy
100%
Cisplatin
75%
Drug Resistance
50%
Polymerase
50%
Malignant Neoplasm
25%
In Vitro
25%
Carcinogenesis
25%
Cancer
25%
Chemotherapy Agent
25%
Lung Cancer Cell Line
25%
Biochemistry, Genetics and Molecular Biology
Cell Death
100%
DNA Damage
100%
Translesion Synthesis
100%
Cisplatin
42%
Drug Resistance
28%
Polymerase
28%
Cancer Cell
14%
Carcinogenesis
14%
Small Molecule
14%